[1] 郗春艳,麻宏伟,华天懿,赵云静.孤独症患儿婴儿时期的行为特征研究[J].中国当代儿科杂志, 2006, 8(6): 470-472.

[2] 李建华, 钟建民, 蔡兰云, 陈勇, 周末芝.三种儿童孤独症行为评定量表临床应用比较[J].中国当代儿科杂志, 2005, 7(2): 59-61.

[3] Molloy CA, Kalkwarf HJ, Manning-Courtney P, Mills JL, Hediger ML. Plasma 25-OH-D concentration in children with autism spectrum disorder[J]. Dev Med Child Neurol, 2010, 52(10): 969-971.

[4] Kanner L. Autistic disturbances of affective contact[J]. Acta Paedopsychiatr, 1968, 35(4): 100-136.

[5] Currenti SA. Understanding and determining the etiology of autism[J]. Cell Mol Neurobiol, 2010, 30(2): 161-171.

[6] Muhle R, Trentacoste SV, Rapin I. The genetics of autism[J]. Pediatrics, 2004, 113(5): e472-e486.

[7] Grant WB, Soles CM. Epidemiologic evidence supporting the role of maternal vitamin D deficiency as a risk factor for the development of infantile autism[J]. Dermatoendocrinology, 2009, 1(4): 223-228.

[8] Cannell JJ. Autism and vitamin D[J]. Med Hypotheses, 2008, 70(4): 750-759.

[9] Holick MF. The vitamin D deficiency pandemic and consequences for nonskeletal health: Mechanisms of action[J]. Mol Aspects Med, 2008, 29(6): 361-368.

[10] Cantorna MT, Zhu Y, Froicu M, Wittke A. Vitamin D status,1,25-dihydroxyvitamin D3, and the immune system[J]. Am J Clin Nutr, 2004, 80(6): 1717S-1720S.

[11] Kesby JP, Eyles DW, Burne TH, McGrath JJ. The effects of vitamin D on brain development and adult brain function[J]. Mol Cell Endocrinol, 2011, 347(1-2): 121-127.

[12] Humble MB, Gustafsson S, Bejerot S. Low serum levels of 25-hydroxyvitamin D (25-OH-D) among psychiatric out-patients in Sweden: Relations with season, age, ethic origin and psychiatric diagnosis[J]. J Steroid Biochem Mol Biol, 2010, 121(1-2): 467-470.

[13] Meguid NA, Hashish AF, Amwar M, Sidhom G. Reduced serum levels of 25-hydroxy and 1,25-dihydroxy vitamin D in Egyptian children with autism[J]. J Altern Complement Med, 2010, 16(6): 641-645.

[14] Gallo S, Jean PS, Rodd C, Weiler HA. Vitamin D supplementation of Canadian infants: practices of Montreal mothers[J]. Appl Physiol Nutr Metab, 2010, 35(3): 303-309.

[15] Herndon AC, DiGuiseppi C, Johnson SL, Leiferman J, Reynolds A.Does nutritional intake differ between children with autism spectrum disorders and children with typical development?[J]. J Autism Dev Disord, 2009, 39(2): 212-222.

[16] Bakare MO, Munir KM. Autism spectrum disorders (ASD) in Africa:a perspective[J]. Afr J Psychiatry, 2011, 14(3): 208-210.

[17] Atladottir HO, Parner ET, Schendel D, Dalsgaard S, Thomsen PH, Thorsen P. Time trends in reported diagnoses of childhood neuropsychiatric disorders:a Danish cohort study[J]. Arch Pediatr Adolesc Med, 2007, 161(2): 193-198.

[18] Lewine JD, Andrews R, Chez M, Patil AA, Devinsky O, Smith M, et al. Magnetoencephalographic patterns of epileptiform activity in children with regressive autism spectrum disorders[J]. Pediatrics, 1999, 104(3): 405-418.

[19] Kalueff AV, Minasyan A, Keisala T, Kuuslahti M, Miettinen S, Tuohimaa P. The vitamin D neuroendocrine system as a target for novel neurotropic drugs[J]. CNS Neurol Disord Drug Targets, 2006, 5(3): 363-371.

[20] McGrath J, Feron F, Eyles D, Mackay SA. Vitamin D:the neglected neurosteroid?[J]. Trends Neurosci, 2001, 24(10): 570-572.

[21] Kocovska E, Fernell E, Billstedt E, Minnis H, Gillberg C. Vitamin D and autism:Clinical review[J]. Res Dev Disabil, 2012, 33(5): 1541-1550.

[22] 崔 健, 陈 虹, 黄秉仁. 维生素D受体最新研究进展[J]. 生理科学进展, 2011, 42(2): 95-99.

[23] Gao L, Tao Y, Zhang L, Jin Q. Vitamin D receptor genetic polymorphisms and tuberculosis: updated systematic review and meta-analysis[J]. Int J Tuberc Lung Dis, 2010, 14(1): 15-23.

[24] Samuel S, Sitrin MD. Vitamin D's role in cell proliferation and differentiation[J]. Nutr Rev, 2008, 66(10 Suppl 2): S116-S124．

[25] Bohnsaek BL, Hirschi KK. Nutrient regulation of cell cycle progression[J]．Annu Rev Nutr, 2004, 24(10): 433-453．

[26] Grant WB, Soles CM. Epidemiologic evidence supporting the role of maternal vitamin D deficiency as a risk factor for the development of infantile autism[J]. Ermatoendocrinol, 2009, 1(4): 223-228.

[27] Evatt ML, DeLong MR, Grant WB, Cannell JJ, Tangpricha V. Autism spectrum disorders following in utero exposure to antiepileptic drugs[J]. Neurology, 2009, 73(12): 997.

[28] Chambers ES, Hawrylowicz CM. The impact of vitamin D on regulatory T cells[J]. Curr Allergy Asthma Rep, 2011, 11(1): 29-36.

[29] Vignali DA, Collison LW, Workman CJ. How regulatory T cells work[J]. Nat Rev Immunol, 2008, 8(7): 523-532.

[30] Bettelli E, Korn T, Oukka M, Kuchroo VK. Induction and effector functions of T(H)17 cells[J]. Nature, 2008, 453(7198): 1051-1057.

[31] Mostafa GA, Shehab AA, Fouad NR. Increased frequency of CD4+CD25high regulatory T cells in the peripheral blood of Egyptian children with autism[J]. J Child Neurol, 2010, 25(3): 328-335.

[32] Jones AP, Tulic MK, Rueter K, Prescott SL. Vitamin D and allergic disease:sunlight at the end of the tunnel?[J].  Nutrients, 2012, 4(1): 13-28.

[33] Zhang HL, Wu J. Role of vitamin D in immune responses and autoimmune diseases, with emphasis on its role in multiple sclerosis[J]. Neurosci Bull, 2010, 26(6): 445-454.

[34] Hamza RT, Awwad KS, Ali MK, Hamed AI. Reduced serum concentrations of 25-hydroxy vitamin D in Egyptian patients with systemic lupus erythematosus: relation to disease activity[J]. Med Sci Monit, 2011, 17(12): CR711-CR718.

[35] Wattjes MP, Doepp F, Bendszus M, Fiehler J. Chronic cerebrospinal venous insufficiency in multiple sclerosis-is multiple sclerosis a disease of the cerebrospinal venous outflow system?[J]. Fortschr Rontgenstr, 2011, 183(6): 523-530.

[36] Mostafa GA, AL-Ayadhi LY. Reduced serum concentrations of 25-hydroxy vitamin D in children with autism: relation to autoimmunity[J]. J Neuroinflammation, 2012, 9: 201-207.

[37] Steck AJ, Stalder AK, Renaud S. Anti-myelin-associated glycoprotein neuropathy[J]. Curr Opin Neurol, 2006, 19(5): 458-463.

[38] Humblea MB, Gustafssonb S, Bejerota S. Low serum levels of 25-hydroxyvitamin D (25-OH-D) among psychiatric out-patients in Sweden:Relations with season,age,ethnic origin and psychiatric diagnosis[J]. J Steroid Biochem Mol Biol, 2010, 121(1-2): 467-470.

[39] Kinney DK, Barch DH, Chayka B, Napoleon S, Munir KM. Environmental risk factors for autism: do they help cause de novo genetic mutations that contribute to the disorder?[J]. Med Hypotheses, 2010, 74(1): 102-106.

[40] Harms LR, BurneTH, Eyles DW, McGrath JJ. Vitamin D and the brain[J]. Best Pract Res Clin Endocrinol Metab, 2011, 25(4): 657-669.

[41] Eyles D, Burne T, McGrath J. Vitamin D in fetal brain development[J]. Semin Cell Dev Biol, 2011, 22(6): 629-636.

[42] Heaney RP, Dowell MS, Hale CA, Bendich A. Calcium absorption varies within the reference range for serum 25-hydroxyvitamin D[J]. J Am Coll Nutr, 2003, 22(2): 142-146.

[43] Bischoff-Ferrari HA, Dietrich T, Orav EJ, Hu FB, Zhang Y, Karlson EW, et al. Higher 25-hydroxyvitamin D concentrations are associated with better lower-extremity function in both active and inactive persons aged > or = 60y[J]. Am J Clin Nutr, 2004, 80(3): 752-758.

[44] Bischoff-Ferrari HA, Giovannucci E, Willett WC, Dietrich T, Dawson-Hughes B. Estimation of optimal serum concentrations of 25-hydroxyvitamin D for multiple health outcomes[J]. Am J Clin Nutr, 2006, 84(1): 18-28.

[45] Cannell JJ, Hollis WB. Use of vitamin D in clinical practice[J].Altern Med Rev, 2008, 13(1): 6-20.

[46] Cannell JJ.On the aetiology of autism[J]. Acta Paediatr, 2010, 99(8): 1128-1130.