About author:: 10.7499/j.issn.1008-8830.2015.08.022
Cite this article:
. Magnetic resonance imaging study on undefined bright objects in the brain tissue of children with neurofibromatosis type 1[J]. CJCP, 2015, 17(8): 873-876.
. Magnetic resonance imaging study on undefined bright objects in the brain tissue of children with neurofibromatosis type 1[J]. CJCP, 2015, 17(8): 873-876.
Ferraz-Filho JR, da Rocha AJ, Muniz MP, et al. Diffusion tensor MR imaging in neurofibromatosis type 1: expanding the knowledge of microstructural brain abnormalities[J]. Pediatr Radiol, 2012, 42(2): 449-454.
[5]
Billiet T, Madler B, Darker, et al. Characterizing the microstructure basis of ""unidentified bright objects"" in neurofibromatosis type 1: a combined in vivo multi-component T2 relaxation and multi-shell diffusion MRI analysis[J]. Neuroimage Clinic, 2014, 4(4): 649-658.
[6]
Khan A, Beri S, Baheerathan A, et al. Globus pallidus high-signal lesions: a predominant MRI finding in children with neurofibromatosis type 1[J]. Ann Indian Acad Neurol, 2013, 16(1): 53-56.
[7]
Sabol Z, Resic B, Gjergja JR, et al. Clinical sensitivity and specificity of multiple T2-hyperintensities on brain magnetic resonance imaging in diagnosis of neurofibromatosis type 1 in children: diagnostic accuracy study[J]. Croat Med J, 2011, 52(4): 488-496.
[8]
Barbier C, Chabernaud C, Barantin L, et al. Proton MR spectroscopic imaging of basal ganglia and thalamus in neurofibromatosis type 1: correlation with T2 hyperintensities[J]. Neuroradiology, 2011, 53(2): 141-148.
[9]
Griffiths PD, Blaser S, Mukonoweshuro W, et al. Neurofibromatosis bright objects in children with neurofibromatosis type 1: a proliferative potential?[J]. Pediatrics, 1999, 104(4): e49.
[10]
Hyman SL, Gill DS, Shores EA, et al. T2 hyperintensities in children with neurofibromatosis type 1 and their relationship to cognitive functioning[J]. J Neurol Neurosurg Psychiatry, 2007, 78(10): 1088-1091.
[11]
Klein-Tasman BP, Janke KM, Luo W, et al. Cognitive and psychosocial phenotype of young children with neurofibromatosis-1[J]. Int Neuropsychol Soc, 2014, 20(1): 88-98.
[12]
Payne JM, Pickering T, Porter M, et al. Longitudinal assessment of cognition and T2-hyperintensities in NF1:an 18-year study[J]. Am J Med Genet A, 2014, 164A(3): 661-665.
[13]
Lehtonen A, Howie E, Trump D, et al. Behaviour in children with neurofibromatosis type 1: cognition, executive function, attention, emotion, and social competence[J]. Dev Med Child Neurol, 2013, 55(2): 111-125.
[14]
Song SK, Sun SW, Ramsbottom MJ, et al. Dysmyelination revealed through MRI as increased radial (but unchanged axial) diffusion of water[J]. Neuroimage, 2002, 17(3): 1429-1436.
[15]
Ford JC, Hackney DB, Lavi E, et al. Dependence of apparent diffusion coefficients on axonal spacing, membrane permeability, and diffusion time in spinal cord white matter[J]. Magn Reson Imaging, 1998, 8(4): 775-782.
[16]
Karlsgodt KH, Rosser T, Lutkenhoff ES, et al. Alterations in white matter microstructure in neurofibromatosis-1[J]. PLoS One, 2012, 7(10): e47854.
