LI Jin, TAO Zhe, AN Ren-Zhe.
Epilepsy as the first presentation in a child with type 1diabetes mellitus[J]. Chinese Journal of Contemporary Pediatrics. 2013, 15(12): 1148-1149 https://doi.org/10.7499/j.issn.1008-8830.2013.12.028
{{custom_sec.title}}
{{custom_sec.title}}
{{custom_sec.content}}
参考文献
[1] 何伟. 糖尿病非酮症高渗综合征合并癫癎持续状态15例临床分析[J].中国糖尿病杂志, 2000, 8(6): 371. [2] 葛文华, 于征淼, 吴智兵. 以癫癎发作为首发症状的糖尿病酮症酸中毒1例报道[J]. 中国现代医生杂志, 2009, 41(7): 139-140. [3] 王有, 闫也. 高血糖导致癫癎单纯部分性发作1例[J]. 长春中医药大学学报, 2008, 24(2): 185. [4] 王华, 郑广群. 小儿癫癎的免疫学进展[J].实用儿科临床杂志, 2011, 26(12): 900-902. [5] Striano P, Errichiello L, Striano S. Autoantibodies to glutamic acid decarboxylase in patients with epilepsy: what is their clinical relevance?[J]. Epilepsy Behav, 2011, 20(1): 145. [6] Saiz A, Blanco Y, Sabater L, Gonzalez F, Bataller L, Casamitjana R, et al. Spectrum of neurological syndromes associated with glutamic acid decarboxylase antibodies: diagnostic clues for this association[J]. Brain, 2008, 131(Pt 10): 2553-2563. [7] Baekkeskov S, Aanstoot HJ, Christgau S, Reetz A, Solimena M, Cascalho M, et al. Identification of the 64K autoantigen in insulin-dependent diabetes as the GABA-synthesizing enzyme glutamic acid decarboxylase[J]. Nature, 1990, 347(6289): 151-156. [8] Tuomi T. Antibodies to glutamic acid decarboxylase reveal latent autoimmune diabetes mellitus in adults with a non-insulin-dependent onset of disease[J]. Diabetes, 1993, 42(2): 359-362. [9] 何君钦, 林丽香, 陈叫钦. 糖尿病患者谷氨酸脱羧酶自身抗体测定的临床意义[J]. 中华内分泌代谢杂志, 1997, 5(2): 97-100. [10] Vincent A, Crino PB. Systemic and neurologic autoimmune disorders associated with seizures or epilepsy[J]. Epilepsia, 2011, 52(3): 12-17. [11] Stagg CJ, Lang B, Best JG, McKnight K, Cavey A, Johansen-Berg H, et al. Autoantibodies to glutamic acid decarboxylase in patients with epilepsy are associated with low cortical GABA levels[J]. Epilepsia, 2010, 51(9): 1898-1901. [12] Vianello M, Bisson G, Dal Maschio M, Vassanelli S, Girardi S, Mucignat C, et al. Increased spontaneous activity of a network of hippocampal neurons in culture caused by suppression of inhibitory potentials mediated by anti-gad antibodies[J]. Autoimmunity, 2008, 41(1): 66-73. [13] Peltola J, Kulmala P, Isojarri J, Saiz A, Latvala K, Palmio J, et al. Autoantibodies to glutamic acid decarboxy lase in patients with therapy-resistant epilepsy[J]. Neurology, 2000, 55(1): 46-50.